William R Roeske

Medicine

Phone(520) 626-6221
Emailwroeske@email.arizona.edu

Source: Scopus
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1 Finished

CELLULAR MECHANISMS IN CARDIAC HYPERTROPY
Goldman, S., Roeske, W. R., Runyan, R. B., White, H., Hartshorne, D., Bahl, J. & Morkin, E.
National Institutes of Health
9/1/77 → 8/31/99
Project: Research project

7962 Citations
48 h-Index
221 Article
12 Review article
3 Chapter
1 Comment/debate
More
- 1 Short survey

Repeated morphine treatment-mediated hyperalgesia, allodynia and spinal glial activation are blocked by co-administration of a selective cannabinoid receptor type-2 agonist
Tumati, S., Largent-Milnes, T. M., Keresztes, A., Ren, J., Roeske, W. R., Vanderah, T. W. & Varga, E. V., Mar 2012, In: Journal of Neuroimmunology. 244, 1-2, p. 23-31 9 p.
Research output: Contribution to journal › Article › peer-review
30 Scopus citations
Tachykinin NK ₁ receptor antagonist co-administration attenuates opioid withdrawal-mediated spinal microglia and astrocyte activation
Tumati, S., Largent-Milnes, T. M., Keresztes, A. I., Yamamoto, T., Vanderah, T. W., Roeske, W. R., Hruby, V. J. & Varga, E. V., Jun 5 2012, In: European Journal of Pharmacology. 684, 1-3, p. 64-70 7 p.
Research output: Contribution to journal › Article › peer-review
23 Scopus citations
Intrathecal PKA-selective siRNA treatment blocks sustained morphine-mediated pain sensitization and antinociceptive tolerance in rats
Tumati, S., Roeske, W. R., Largent-Milnes, T. M., Vanderah, T. W. & Varga, E. V., Jul 15 2011, In: Journal of Neuroscience Methods. 199, 1, p. 62-68 7 p.
Research output: Contribution to journal › Article › peer-review
14 Scopus citations
Sustained morphine-mediated pain sensitization and antinociceptive tolerance are blocked by intrathecal treatment with Raf-1-selective siRNA
Tumati, S., Roeske, W. R., Largent-Milnes, T., Wang, R., Vanderah, T. W. & Varga, E. V., Sep 2010, In: British Journal of Pharmacology. 161, 1, p. 51-64 14 p.
Research output: Contribution to journal › Article › peer-review
13 Scopus citations
Sustained morphine treatment augments prostaglandin E₂-evoked calcitonin gene-related peptide release from primary sensory neurons in a PKA-dependent manner
Tumati, S., Roeske, W. R., Vanderah, T. W. & Varga, E. V., Dec 1 2010, In: European Journal of Pharmacology. 648, 1-3, p. 95-101 7 p.
Research output: Contribution to journal › Article › peer-review
12 Scopus citations

CELLULAR MECHANISMS IN CARDIAC HYPERTROPY

Repeated morphine treatment-mediated hyperalgesia, allodynia and spinal glial activation are blocked by co-administration of a selective cannabinoid receptor type-2 agonist

Tachykinin NK 1 receptor antagonist co-administration attenuates opioid withdrawal-mediated spinal microglia and astrocyte activation

Intrathecal PKA-selective siRNA treatment blocks sustained morphine-mediated pain sensitization and antinociceptive tolerance in rats

Sustained morphine-mediated pain sensitization and antinociceptive tolerance are blocked by intrathecal treatment with Raf-1-selective siRNA

Sustained morphine treatment augments prostaglandin E2-evoked calcitonin gene-related peptide release from primary sensory neurons in a PKA-dependent manner

Tachykinin NK ₁ receptor antagonist co-administration attenuates opioid withdrawal-mediated spinal microglia and astrocyte activation

Sustained morphine treatment augments prostaglandin E₂-evoked calcitonin gene-related peptide release from primary sensory neurons in a PKA-dependent manner