Loss of Spontaneous Blinking in a Patient With Balint's Syndrome

Robert T. Watson, Steven Z. Rapcsak

Research output: Contribution to journalArticle

11 Scopus citations

Abstract

A patient with Balint's syndrome caused by bilateral parieto-occipital lesions lost spontaneous blinking, suggesting that humans, like nonhuman primates, have parietal lobe neurons that are important for blinking. Although the functions of spontaneous blinking are not known, they may help initiate some saccades and, like saccades, be involved in the cancellation of thalamic inhibitory postsynaptic potentials, thereby facilitating processing of new foveal targets. Spontaneous blinking may also facilitate sensory relay during sustained attention and, therefore, help prevent fading of a retinal image.

Original languageEnglish (US)
Pages (from-to)567-570
Number of pages4
JournalArchives of Neurology
Volume46
Issue number5
DOIs
StatePublished - May 1989

ASJC Scopus subject areas

  • Arts and Humanities (miscellaneous)
  • Clinical Neurology

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