Abstract
The genetic mutation in cystic fibrosis causes a generalised dysfunction of the exocrine system. The subsequent increase in energy needs due to pulmonary infections, increased basal metabolic rate, and malabsorption are not compensated for by increased food intake, hence, malnutrition becomes a problem for most patients. Biological control mechanisms for body energy regulation may be impaired, causing anorexia directly and contributing to learned behaviors which further decrease intake. An interdisciplinary approach to treatment is suggested which would include behavioral, dietary, and medical components. Early and continuous assessment and modification of problematic eating behaviors and related environmental factors along with nutrition education may delay the need for artificial supplementation, and augment their use by increasing oral intake and compliance with procedures. Anxiety due to increasing disease severity may contribute to anorexia and treatment noncompliance. Behavioral training in identification of Stressors and relaxation techniques may be helpful in reducing stress.
| Original language | English (US) |
|---|---|
| Pages (from-to) | 315-331 |
| Number of pages | 17 |
| Journal | Clinical Psychology Review |
| Volume | 11 |
| Issue number | 3 |
| DOIs | |
| State | Published - 1991 |
| Externally published | Yes |
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ASJC Scopus subject areas
- Psychiatry and Mental health
- Clinical Psychology
Cite this
Malnutrition in cystic fibrosis : A behavioral conceptualization of cause and treatment. / Bowen, Anne Markey; Stark, Lori J.
In: Clinical Psychology Review, Vol. 11, No. 3, 1991, p. 315-331.Research output: Contribution to journal › Article
}
TY - JOUR
T1 - Malnutrition in cystic fibrosis
T2 - A behavioral conceptualization of cause and treatment
AU - Bowen, Anne Markey
AU - Stark, Lori J.
PY - 1991
Y1 - 1991
N2 - The genetic mutation in cystic fibrosis causes a generalised dysfunction of the exocrine system. The subsequent increase in energy needs due to pulmonary infections, increased basal metabolic rate, and malabsorption are not compensated for by increased food intake, hence, malnutrition becomes a problem for most patients. Biological control mechanisms for body energy regulation may be impaired, causing anorexia directly and contributing to learned behaviors which further decrease intake. An interdisciplinary approach to treatment is suggested which would include behavioral, dietary, and medical components. Early and continuous assessment and modification of problematic eating behaviors and related environmental factors along with nutrition education may delay the need for artificial supplementation, and augment their use by increasing oral intake and compliance with procedures. Anxiety due to increasing disease severity may contribute to anorexia and treatment noncompliance. Behavioral training in identification of Stressors and relaxation techniques may be helpful in reducing stress.
AB - The genetic mutation in cystic fibrosis causes a generalised dysfunction of the exocrine system. The subsequent increase in energy needs due to pulmonary infections, increased basal metabolic rate, and malabsorption are not compensated for by increased food intake, hence, malnutrition becomes a problem for most patients. Biological control mechanisms for body energy regulation may be impaired, causing anorexia directly and contributing to learned behaviors which further decrease intake. An interdisciplinary approach to treatment is suggested which would include behavioral, dietary, and medical components. Early and continuous assessment and modification of problematic eating behaviors and related environmental factors along with nutrition education may delay the need for artificial supplementation, and augment their use by increasing oral intake and compliance with procedures. Anxiety due to increasing disease severity may contribute to anorexia and treatment noncompliance. Behavioral training in identification of Stressors and relaxation techniques may be helpful in reducing stress.
UR - http://www.scopus.com/inward/record.url?scp=0025876583&partnerID=8YFLogxK
UR - http://www.scopus.com/inward/citedby.url?scp=0025876583&partnerID=8YFLogxK
U2 - 10.1016/0272-7358(91)90106-5
DO - 10.1016/0272-7358(91)90106-5
M3 - Article
AN - SCOPUS:0025876583
VL - 11
SP - 315
EP - 331
JO - Clinical Psychology Review
JF - Clinical Psychology Review
SN - 0272-7358
IS - 3
ER -