Of flaky tails and itchy skin

Research output: Contribution to journalArticle

13 Citations (Scopus)

Abstract

A new study defines the flaky tail mouse as a model for human atopic dermatitis caused by a null mutation in the gene encoding filaggrin, a key component of the epidermal barrier. Research in these mice will help explain how a disrupted barrier contributes to the pathogenesis of atopic dermatitis and to asthma arising in the context of atopic skin disease.

Original languageEnglish (US)
Pages (from-to)512-513
Number of pages2
JournalNature Genetics
Volume41
Issue number5
DOIs
StatePublished - May 2009

Fingerprint

Atopic Dermatitis
Tail
Skin
Skin Diseases
Asthma
Mutation
Research
Genes
filaggrin

ASJC Scopus subject areas

  • Genetics

Cite this

Of flaky tails and itchy skin. / Vercelli, Donata.

In: Nature Genetics, Vol. 41, No. 5, 05.2009, p. 512-513.

Research output: Contribution to journalArticle

Vercelli, Donata. / Of flaky tails and itchy skin. In: Nature Genetics. 2009 ; Vol. 41, No. 5. pp. 512-513.
@article{f248e9225aee4625b58eae3d035af65d,
title = "Of flaky tails and itchy skin",
abstract = "A new study defines the flaky tail mouse as a model for human atopic dermatitis caused by a null mutation in the gene encoding filaggrin, a key component of the epidermal barrier. Research in these mice will help explain how a disrupted barrier contributes to the pathogenesis of atopic dermatitis and to asthma arising in the context of atopic skin disease.",
author = "Donata Vercelli",
year = "2009",
month = "5",
doi = "10.1038/ng0509-512",
language = "English (US)",
volume = "41",
pages = "512--513",
journal = "Nature Genetics",
issn = "1061-4036",
publisher = "Nature Publishing Group",
number = "5",

}

TY - JOUR

T1 - Of flaky tails and itchy skin

AU - Vercelli, Donata

PY - 2009/5

Y1 - 2009/5

N2 - A new study defines the flaky tail mouse as a model for human atopic dermatitis caused by a null mutation in the gene encoding filaggrin, a key component of the epidermal barrier. Research in these mice will help explain how a disrupted barrier contributes to the pathogenesis of atopic dermatitis and to asthma arising in the context of atopic skin disease.

AB - A new study defines the flaky tail mouse as a model for human atopic dermatitis caused by a null mutation in the gene encoding filaggrin, a key component of the epidermal barrier. Research in these mice will help explain how a disrupted barrier contributes to the pathogenesis of atopic dermatitis and to asthma arising in the context of atopic skin disease.

UR - http://www.scopus.com/inward/record.url?scp=66749150976&partnerID=8YFLogxK

UR - http://www.scopus.com/inward/citedby.url?scp=66749150976&partnerID=8YFLogxK

U2 - 10.1038/ng0509-512

DO - 10.1038/ng0509-512

M3 - Article

VL - 41

SP - 512

EP - 513

JO - Nature Genetics

JF - Nature Genetics

SN - 1061-4036

IS - 5

ER -