Point: Counterpoint "Release of an endothelium-derived vasoconstrictor and RhoA/Rho kinase-mediated calcium sensitization of smooth muscle cell contraction are/are not the main effectors for full and sustained hypoxic pulmonary vasoconstriction".

Research output: Contribution to journalArticle

Original languageEnglish (US)
JournalJournal of Applied Physiology
Volume102
Issue number5
StatePublished - May 2007
Externally publishedYes

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Intracellular Signaling Peptides and Proteins
rhoA GTP-Binding Protein
rho-Associated Kinases
Calcium Signaling
Protein-Serine-Threonine Kinases
Endothelins
Vascular Endothelium
Vasoconstrictor Agents
Endothelin-1
Muscle Contraction
Vasoconstriction
Vascular Smooth Muscle
Pulmonary Artery
Smooth Muscle Myocytes
Endothelium
Calcium
Lung
Hypoxia

ASJC Scopus subject areas

  • Physiology
  • Endocrinology
  • Orthopedics and Sports Medicine
  • Physical Therapy, Sports Therapy and Rehabilitation

Cite this

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title = "Point: Counterpoint {"}Release of an endothelium-derived vasoconstrictor and RhoA/Rho kinase-mediated calcium sensitization of smooth muscle cell contraction are/are not the main effectors for full and sustained hypoxic pulmonary vasoconstriction{"}.",
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