TRPV1 channels

Not so inactive on the ER

Elliot Imler, Konrad E Zinsmaier

Research output: Contribution to journalArticle

1 Citation (Scopus)

Abstract

In this issue of Neuron, Wong etal. (2014) report a remarkable evolutionarily conserved role for the Drosophila TRPV1 homolog Inactive controlling synaptic growth at larval neuromuscular junctions by facilitating Ca2+ release from the endoplasmic reticulum. In this issue of Neuron, Wong etal. (2014) report a remarkable evolutionarily conserved role for the Drosophila TRPV1 homolog Inactive controlling synaptic growth at larval neuromuscular junctions by facilitating Ca2+ release from the endoplasmic reticulum.

Original languageEnglish (US)
Pages (from-to)659-661
Number of pages3
JournalNeuron
Volume84
Issue number4
DOIs
StatePublished - Nov 19 2014

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Neuromuscular Junction
Endoplasmic Reticulum
Drosophila
Neurons
Growth

ASJC Scopus subject areas

  • Neuroscience(all)
  • Medicine(all)

Cite this

TRPV1 channels : Not so inactive on the ER. / Imler, Elliot; Zinsmaier, Konrad E.

In: Neuron, Vol. 84, No. 4, 19.11.2014, p. 659-661.

Research output: Contribution to journalArticle

Imler, Elliot ; Zinsmaier, Konrad E. / TRPV1 channels : Not so inactive on the ER. In: Neuron. 2014 ; Vol. 84, No. 4. pp. 659-661.
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